- thjuland
- Sex: M
- Data Quality: 3 stars
- MS: 49 yrs
- Type: Relapsing-Remitting
- Sensation: Moderate
- Overall: Moderate
- Cognition: moderate
- Vision: mild
- Speech: mild
- Swallowing: moderate
- Upper limb: moderate
- Walking: moderate
- thjuland
- Male, 61 years
- Albany, NY
More About thjuland
Hi folks,
The below information is the report from my 3rd MRI, which finally gave me a confirmed MS diagnosis. Over the previous 14 years, I had gone from 'possible MS', to 'benign MS' and I am now finally just another MSer--person living with MS, rather than being only a patient. :-)
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Albany Medical Center Hospital
Radiological Consultation
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Name: Copeland, Thomas DOB: 21-Mar-1951 Sex: M
Date: 08-Apr-1995 MRN: 1347419 Ward: OP
Clinical: Evaluation For Multiple Sclerosis.
Exam: MRI Brain
Impression:
Numerous white matter lesions within both cerebral hemispheres,
the corpus callosum, the brainstem and cerebellum, consistent
with the clinical diagnosis of multiple sclerosis. Although the
previous MRI from 7-06-88 is not available, there is mention on
the report of white matter pathology which was suspicious for
demyelinating disease.
Technical Factors: 1.5 Telsa magnetic imaging of the brain was performed with sagittal T1, axial T1 and axial dual echo T2 images. Sagittal T2 weighted images.
1 - There are numerous focal lesions withn the periventricular white matter of both
cerebral hemispheres as well as focal abnormality within the corpus callosum
and the forcep major bilaterally.
2 - A 9mm plaque is located in the posterolateral aspect of the left side of the pons
just ventral to the middle cerebellar peduncle.
3 - Questionable punctate lesion is identified in the left lateral cerebellar hemisphere.
4 - The punctate lesions are identified within the superior white matter of both
cerebral hemispheres and in the right forceps minor.
5 - On the sagittal T2 weighted images, small lesions are noted within the corpus
callosum.
6 - Another 6mm lesion is identified within the right basal ganglia white matter.
7 - The ventricles and the vascular flow voids are normal. There is no associated
hemorrhage or mass effect.
signed: WAW., M.D.
A-70 NL. MD
Albany Medical College 10-Apr-1995 12:19
Albany, NY 12208 102462165
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6th MRI
DOS: 11-11-2005 Patient: Copeland, Thomas
Daniel J Silverman, MD Soc. Sec. # 083-44-5167
314 S Manning Blvd. DOB: 3-21-1951
Albany, NY 12208 Account # 883
70553 MR Brain WO/W Contrast
Clinical History: Multiple Sclerosis
Findings:
Using a 1.0 Tesla superconducting magnet, T1 weighted and FLAIR sagittal, and T1 weighted, T2 weighted, FLAIR and diffusion-weighted axial images were obtained. Following intravenous administration of gadoteridol contrast, T1 weighted axial images were again obtained.
Comparison is made with previous MRI of the brain obtained on 5-26-2004.
The ventricles are at the upper limits of normal in size or slightly enlarged. There is focal atrophy in the posterior parietal lobes bilaterally. These findings are unchanged compared to previous study. (View: My MRI image)
Multiple oval areas of increased signal are noted on FLAIR and T2 weighted images in the white matter of both cerebral hemispheres. (View: Brain MRI)
Since the previous MRI scan, a 6-7mm area of hyperintensity has developed in the ventral pons to the right of midline, seen on FLAIR and T2 weighted images, with restricted diffusion.
There is no evidence of abnormal contrast enhancement associated with the right ventral pontine abnormality.
New punctate T2 signal abnormalities are noted in the left cerebellar hemisphere and in the periventricular white matter of the left parietal lobe.
There is linear enhancement in the left parietal lobe following gadoteridol contrast administration, and there may be minimal rounded enhancement adjacent to the body of the left lateral ventricle as well.
These areas of enhancement are not tightly correlated with areas of T2 abnormality. There is no evidence of mass effect or extracerebral fluid collection.
There is atrophy of the corpus callosum (View MRI image).
Impression:
1 - Mild atrophy and white matter signal changes in both cerebral hemispheres,
compatible with the clinical diagnosis of Multiple Sclerosis.
2 - A 6-7mm area of T2 hyperintensity with restricted diffusion has developed
in the ventral aspect of the pons to the right of midline. This may well
represent a recent, active plaque.
3 - Linear area of signal enhancement in the left parietal white matter. While
this is not tightly correlated with signal abnormality in this location on
FLAIR and T2 weighted sequences, this could represent an additional area
of acute demyelination.
4 - Stable focal atrophy involving both posterior parietal lobes, left more
than right (View: My MRI image).
Dictated by: S S, MD.
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