thjuland
thjuland
Sex: M
Data Quality: 3 stars
MS: 49 yrs
Type: Relapsing-Remitting
Sensation: Moderate
Overall: Moderate
Cognition: moderate
Vision: mild
Speech: mild
Swallowing: moderate
Upper limb: moderate
Walking: moderate
thjuland
Male, 61 years
Albany, NY
Primary Condition
MS and 4 more
Type
Relapsing-Remitting
First symptom
Sep 1962
Diagnosis
Sep 1981

About thjuland

Although my official MS onset was in 1981; it actually began in 1962 when I first had subclinical ON, but I was too young and so not told. After my diagnosis my ophthalmologist explained it. I worked until 1992, when I had surgery, then more lesions and I became disabled. Most of my deficits are invisible, being mostly cognitive and I still continue to walk, generally appear normal but aren't. Most of my time is spent online, researching MS material and participating in online MS support groups. I maintain a website, totally dedicated to understanding MS -> http://www.thjuland.net/

Profile Activity
22714 Views
Member since: Jun 23, 2007 Last Login May 26, 2012

Other Conditions

  1. Asthma
    First symptom
    Diagnosis
  2. GERD (Gastroesophageal reflux disease)
    First symptom
    Diagnosis
  3. Pneumonia
    First symptom
    ?
    Diagnosis
    ?
    Stopped
    Stopped Reason
    I never had this.
  4. Vasovagal Syncope
    First symptom
    Diagnosis

More About thjuland

Hi folks,
The below information is the report from my 3rd MRI, which finally gave me a confirmed MS diagnosis. Over the previous 14 years, I had gone from 'possible MS', to 'benign MS' and I am now finally just another MSer--person living with MS, rather than being only a patient. :-)

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                      Albany Medical Center Hospital
                               Radiological Consultation

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Name: Copeland, Thomas        DOB: 21-Mar-1951    Sex: M
Date: 08-Apr-1995        MRN: 1347419        Ward: OP

Clinical: Evaluation For Multiple Sclerosis.

Exam: MRI Brain


Impression:
    Numerous white matter lesions within both cerebral hemispheres,
    the corpus callosum, the brainstem and cerebellum, consistent
    with the clinical diagnosis of multiple sclerosis. Although the
    previous MRI from 7-06-88 is not available, there is mention on
    the report of white matter pathology which was suspicious for
    demyelinating disease.

Technical Factors: 1.5 Telsa magnetic imaging of the brain was performed with sagittal T1, axial T1 and axial dual echo T2 images. Sagittal T2 weighted images.

1 - There are numerous focal lesions withn the periventricular white matter of both
     cerebral hemispheres as well as focal abnormality within the corpus callosum
     and the forcep major bilaterally.
2 - A 9mm plaque is located in the posterolateral aspect of the left side of the pons
      just ventral to the middle cerebellar peduncle.
3 - Questionable punctate lesion is identified in the left lateral cerebellar hemisphere.
4 - The punctate lesions are identified within the superior white matter of both
      cerebral hemispheres and in the right forceps minor.
5 - On the sagittal T2 weighted images, small lesions are noted within the corpus
      callosum.
6 - Another 6mm lesion is identified within the right basal ganglia white matter.
7 - The ventricles and the vascular flow voids are normal. There is no associated
      hemorrhage or mass effect.

            signed: WAW., M.D.
A-70 NL. MD
Albany Medical College                10-Apr-1995 12:19
Albany, NY 12208                       102462165

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                                                                                     6th MRI

DOS: 11-11-2005                    Patient: Copeland, Thomas
Daniel J Silverman, MD          Soc. Sec. # 083-44-5167
314 S Manning Blvd.               DOB: 3-21-1951
Albany, NY 12208                  Account # 883

70553 MR Brain WO/W Contrast
Clinical History:  Multiple Sclerosis

Findings:
Using a 1.0 Tesla superconducting magnet, T1 weighted and FLAIR sagittal, and T1 weighted, T2 weighted, FLAIR and diffusion-weighted axial images were obtained. Following intravenous administration of gadoteridol contrast, T1 weighted axial images were again obtained.

Comparison is made with previous MRI of the brain obtained on 5-26-2004.

The ventricles are at the upper limits of normal in size or slightly enlarged. There is focal atrophy in the posterior parietal lobes bilaterally. These findings are unchanged compared to previous study. (View: My MRI image)

Multiple oval areas of increased signal are noted on FLAIR and T2 weighted images in the white matter of both cerebral hemispheres. (View: Brain MRI)

Since the previous MRI scan, a 6-7mm area of hyperintensity has developed in the ventral pons to the right of midline, seen on FLAIR and T2 weighted images, with restricted diffusion.

There is no evidence of abnormal contrast enhancement associated with the right ventral pontine abnormality.

New punctate T2 signal abnormalities are noted in the left cerebellar hemisphere and in the periventricular white matter of the left parietal lobe.

There is linear enhancement in the left parietal lobe following gadoteridol contrast administration, and there may be minimal rounded enhancement adjacent to the body of the left lateral ventricle as well.

These areas of enhancement are not tightly correlated with areas of T2 abnormality. There is no evidence of mass effect or extracerebral fluid collection.

There is atrophy of the corpus callosum (View MRI image).

Impression:
1 - Mild atrophy and white matter signal changes in both cerebral hemispheres,
      compatible with the clinical diagnosis of Multiple Sclerosis.

2 - A 6-7mm area of T2 hyperintensity with restricted diffusion has developed
      in the ventral aspect of the pons to the right of midline. This may well
      represent a recent, active plaque.

3 - Linear area of signal enhancement in the left parietal white matter. While
      this is not tightly correlated with signal abnormality in this location on
      FLAIR and T2 weighted sequences, this could represent an additional area
      of acute demyelination.

4 - Stable focal atrophy involving both posterior parietal lobes, left more
      than right (View: My MRI image).

Dictated by: S S, MD.

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